Hi Simply Southern,
Its hard to know what to make of Crohns Disease SNPs.
Of the ones you listed I had 8 for which CD risk was normal and 8 with slightly raised risk of CD.
Of the total of 149 CD SNPs I had 99 - Not set; 23 of Good Repute and 27 of Bad Repute.
Of the CD SNPs of Bad Repute the most interesting were those at the NOD2 locus on chromosome 16 since the NOD2 locus is considered to represent one of the strongest genetic risk factors for Crohns Disease.
They were
rs2066843 (T;T) 4.1X risk of CD
rs17221417 (G;G) 1.9X risk of CD and
rs2076756 (G;G) 1.7X risk of CD.

I don't think there are any articles which put a simple hypothesis ERAP1 bad repute SNPs -> axial disease Vs IL23R bad repute SNPs -> peripheral diseae.

You might have been thinking of this article - which is quite dated now.
http://www.sciencecodex.com/major_geneti..._treatment_hope

Understandably, most of the AS cohorts in genetic studies have in the past enrolled probably mainly classic AS cases - and very few more atypical cases - like those with an undifferientiaed spondyloarthropthy.

Recently the focus has been on IL23 and IL23R because of the discovery of a population of entheseal-resident IL23R+ T cells. It is speculated that other unique IL23R+ T cells might populate the skin, the gut, and the eyes.

Dx Oct 2006 B27+ undifferentiated spondlyarthropathy (uSpA) with mild sebhorrhoeic dermatitis and mild Inflammatory Bowel Disease (IBD) controlled by NSD since 2007.