This is not a theory; it is the observation that every case thus far studied has been caused by a single pathogen
does this include the studies where large percentages of AS patients had no culturable klebsiella in stool, no raised antibodies to klebsiella, and there was no difference in disease activity between klebsiella positive and klebsiella negative patients?
NONSENSE! You might want to re-read the study and pay attention this time. There may be a quiz at the end. Hint: It is still the “Etiopathogenesis…” paper.
i just reread the 'The Use of a Low Starch Diet in the Treatment of Patients Suffering from Ankylosing Spondylitis' paper and could not find any mention of controlling for any dietary variables or even measuring starch intake. can you provide a link to the “Etiopathogenesis…” paper or provide some quotes from it?
Molecular mimicry fully explains the gender differences both in frequency as well as presentation, and also why some patients experience iritis while others do not, which is the same question as variation in sites of disease activity. It also offers the best explanation as to why some individuals fuse and others do not.
would you care to elaborate on how it explains gender differences and fusing.
Boy-HOWDY, am I glad I edited my original response to You!
i happened to read the original before admin made you change it. i actually thought it was some of the more creative vitriolic prose that you have come up with and i would have been happy for it to stay as the original version.
But You have taken this statement so far from its original context that it must at least be speciously true...for You alone.
i plead guilty to the charge of taking the quote out of context.
The scientific community agrees with Ebringer about three-to-one
can you provide references or some other way to back up that statement other than "i just made it up".
It will either stand up to this level of scrutiny or it will not. I am taking bets now—care to make a donation, er I mean wager?!
i would love to make a wager. if Ebringer's klebsiella molecular mimicry theory as the cause of all cases of AS ever makes the transition from speculative theory to scientific fact, i will run naked from new york to los angeles wearing a sign around my neck saying 'i was wrong, all hail lord dragonslayer'. what will you be putting up for your half of the wager?
No, although I was aware of this fact, it is not relevant to the primary question: Do bowel or even other germs cause AS? I resolved to my own satisfaction that a) A Gram negative germ is involved and b) That pathogen is most likely one that has already been suspected and c) Hijacks (monopolizes) the secondary immune systems of all AS patients ("all" thus far studied).
as you may have noticed, in this thread i have repeatedly stated that i am arguing against claims that "all cases of AS are caused by klebsiella via molecular mimicry". if you want to claim that AS is caused by "bowel or even other germs" then i am in complete agreement. i would even join hands around the campfire for a rousing chorus of kumbaya.
