Hi Bilko... I
promise I'm not trying to frustrate you, but would really like to hear more on your point of view.
In reply to:
If you had inflammation and pain but no elevated klebs IgA then it wasn't AS. Otherwise it was.
What if Ebringer's premise of a single trigger/causal mechanism fails to envelope an different group of real AS cases? What if there IS another group (as Wally theorises) who are non-responsive to diet, and test negative for elevated kleb IgA, but DO in fact have actual AS? It would certainly explain why diet worked in all of Ebringer's patients, and would also explain why some don't discern a benefit.
This jumped to mind, since most clinicians don't use this specific test in determining an AS dx. Certainly their failure to test for this could simply reflect their failure to adopt his theory as a whole, but this in itself doesn't speak to or rule out a 2nd causal possibility... true? So I guess my real question is... would Dr. Ebringer have ruled out someone with SI fusion, iritis, and all the classic symptoms due to a neg IgA, since it didn't fit the theory? Is the idea of 2 different initiating events resulting in the same affliction.. beyond the realm of possibility?
Your insight is appreciated!
mig
